ILLNESS of HUMAN
That we know about illnesses
Tuesday, 22 May 2012
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The length of capillaries in a kidney makes about 25 kilometers. The filtrational surface of a kidney reaches 1,5 square meters.
VARICOSE PHLEBECTASIA OF LEGS

Varicose phlebectasia of legs (varicose illness) - expansion of the hypodermic (superficial) veins, accompanied an incompetence of valves and disturbance of outflow of a venous blood. Primary varicose expansion is bound to delicacy of a venous wall and functional disturbances of the valval apparatus of veins. Pregnancy, an obesity, long stay in position costing, congenital delicacy of a connecting tissue, wearing a stocking with hard elastics promote development of disease. Secondary varicose expansion results from disturbance of venous outflow, for example at tumours, traumas. Physiology of a venous circulation of an extremity. The current of a venous blood to heart is provided with reduction of muscles of an anticnemion and a femur (a is muscular-venous pomp) and a pulsation of arteries. At reduction of muscles of an anticnemion and a femur there is a prelum of venous sine, the blood from them acts in the deep veins of an extremity equipped by valves that causes the referred venous blood flow in proximal departments of an extremity and a vein of a basin. In a phase of a relaxation of muscles under condition of a solvency of valves of deep veins there is no return of a blood from veins of a basin, and in deep veins the blood from superficial system through veins and from system of muscles of an anticnemion acts. At a varicose phlebectasia rising of pressure in veins leads to a failure of valves of veins therefore at muscular reductions the blood under greater pressure acts from deep system in superficial. There is the local venous hypertensia more expressed in the bottom third of an anticnemion, where veins the most powerful. Rising of pressure in superficial veins leads to their expansion. At the same time rising of pressure in a venous department of microcirculation leads to occurrence of edemas and an emigration of erythrocytes (at long rising pressure the induration and a xanthopathy are marked), and also opening of arteriovenous shunts. It causes appreciable depression of a blood flow in capillaries, decrease of perfusion, a hypoxia of tissues and occurrence of ulcers.

Signs, current. The basic sign in the beginning of disease - a varicose phlebectasia; at indemnification of a venous circulation of other complaints can and to not be. In process of advance of disease there is a fatigability, feeling of gravity in legs, cramps in muscles of legs, especially by the evening, and sometimes and at night. Edemas usually arise by the evening, especially after long standing, after night rest edemas completely disappear. In due course alongside with advance of a varicose phlebectasia there are trophic disturbances, more often localized on an internal surface of the bottom third of anticnemion: an induration, a pigmentation, a dermatitis, then - the trophic ulcer, badly giving in to treatment.

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